Genes and Lifelong Sexlessness

Lifelong sexlessness (virginity past 30) has a measurable, moderate genetic component: twin data imply about 25% MZ concordance for men and ~20% for women, and top polygenic predispositions predict substantially higher risk. The genetics are largely polygenic common‑variant effects and differ between sexes, while the modern mating environment amplifies who ends up in the sexless tail. — This reframes the 'blackpill' debate from a binary genetic determinism versus pure social explanation into a mixed, sex‑specific genetics × environment question with implications for policy, public health, and social narratives about masculinity and dating inequality.